Helicobacter pylori, is the causative agent of gastritis, gastric ulcers, gastric cancer and mucosal-associated lymphoid tissue tumor (MALT). There are several treatment schemes for the eradication of this bacterium.
The CDH1 gene, coding for the E-cadherin protein, is linked to gastric cancer (GC) susceptibility and tumor invasion. The human epidermal growth factor receptor 2 (HER2) is amplified and overexpressed in a portion of GC.
The knowledge about carcinogenesis and the molecular events that lead to cell transformation in gastric cancer (GC) has been a challenge to the scientific community. In this way, studies involving small non-coding RNAs have been widely explored.
Helicobacter pylori (Hp) is the main bacterial agent in gastroduodenal diseases.
Gastric carcinogenesis, in the form of intestinal-type gastric cancer (GC), consists of a series of sequential lesions known as Correa's multistep cascade of gastric cancer.
Gastric cancer is the fifth commonest incident malignancy represents 6.8% of the cancers (952,000 cases/2012)and it is the third leading cause of death by cancer for both genders worldwide 8.8% (723,000 deaths), Epidemiologic studies have shown the positive impact of the Mediterranean Diet.
